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"The sections of DNA found at the ends of chromosomes are known as telomeres, and the shortening of telomeres is considered an indicator of the aging process—the faster the shortening, the faster the aging. The study found that higher D concentrations correlate to longer telomeres and a reduced aging process. Interestingly, osteoporosis, another vitamin D-deficiency disease, also correlates closely to shortened telomeres."
- Marc Sorenson, Solar Power For Optimal Health (Get the book.)

"Wouldn't it be interesting, asked some gene researchers, if we could estimate longevity based on the length of a person's telomeres? It seemed a sensible theory; longer telomeres would mean longer-lived cells, and longer-lived cells would, hypothetically, mean longer life for the organism. When studies were carried out on mice, however, the theory didn't hold up: Strains of mice with long telomeres didn't live any longer than those with average-length telomeres. There are other holes in the argument that our telomeres decide our life span."
- David Heber, M.D., Ph.D., What Color is Your Diet? (Get the book.)

"The study found that higher D concentrations correlate to longer telomeres and a reduced aging process. Interestingly, osteoporosis, another vitamin D-deficiency disease, also correlates closely to shortened telomeres.96 Considering the above information, especially when added to the striking protective effects of vitamin D in reducing the major degenerative and infectious diseases already discussed, it is not surprising that research conducted in August of 2008 showed that people with vitamin D levels below 17."
- Marc Sorenson, Solar Power For Optimal Health (Get the book.)

"Today, scientists are furthering our understanding of this process of cell senescence by taking note of structures called telomeres; these are small fragments, composed of DNA, that form the protective ends of chromosomes. What scientists are seeing is that with each new cell division, the telomeres are shortened, and that after these structures shrink down to a certain size, cell division stops. In other words, telomeres are a kind of biological clock, with decreasing length indicating a decreasing amount of cell lifetime left. But what if we could replenish shrinking telomeres?"
- Gary Null, Gary Null's Power Aging (Get the book.)

"In other words, there must be an increase in the brightness of the telomeres (chromosomal tips). Without these biophoton emissions, any product or supplement is useless, because it requires life to regenerate and sustain life. Love is all there is. Life is love in action. Collecting Primal Energy Certain primary source (unrefined) minerals, especially quartz crystals, are natural concentrators of the primal ullf (biophotons). Crystals can be programmed, just like microchips, to hold and radiate the magnetic love energy frequency."
- Ron Garner, Conscious Health: A Complete Guide to Wellness Through Natural Means (Get the book.)

"First discovered by a group of scientists at the Geron Corporation in Menlo Park, California, telomeres are sequences of nucleic acids extending from the ends of chromosomes. telomeres act to maintain the integrity of our chromosomes. Every time our cells divide, telomeres are shortened, leading to cellular damage and cellular death associated with aging. Scientists discovered that the key element in rebuilding our disappearing telomeres is the "immortalizing" enzyme telomerase—an enzyme found only in germ cells and cancer cells."
- Ronald Klatz and Robert Goldman, Stopping the Clock: Longevity for the New Millenium (Get the book.)

"Atherosclerosis 38: 417-21, 1981] J Chromosomes have end caps called telomeres. Telomerase is an enzyme that repairs broken telomeres and extends the life of cells. Since cancer cells are immortal, the inhibition of telomerase could induce cell death and combat a widely variety of cancers. Allicin effectively inhibits telomerase, blocks the division of tumor cells and can be used to treat cancer. In lab studies, allicin reduces tumor incidence and survival is prolonged. Allicin induces apoptosis, the slow die off of tumor cells."
- Bill Sardi, You Don't Have to be Afraid of Cancer Anymore (Get the book.)

"It seemed a sensible theory; longer telomeres would mean longer-lived cells, and longer-lived cells would, hypothetically, mean longer life for the organism. When studies were carried out on mice, however, the theory didn't hold up: Strains of mice with long telomeres didn't live any longer than those with average-length telomeres. There are other holes in the argument that our telomeres decide our life span."
- David Heber, M.D., Ph.D., What Color is Your Diet? (Get the book.)

"All cells have telomeres, short strips of DNA, but only cancer cells and sperm cells have telomerase. When a cell is young, it contains a chain of more than 1,000 telomeres. Each time the cell divides, it uses up ten or twenty telomeres from the chain. Eventually, the chain is devoured and the cell loses its ability to reproduce. The chain of telomeres in cancer cells is not exhausted. The length of the chain is thought to be protected by the enzyme telomerase. Scientists believe that telomerase enables the mutated cell to outwit death."
- Laurie Deutsch Mozian, M.S., R.D., Foods That Fight Disease: A Simple Guide to Using and Understanding Phytonutrients to Protect and Enhance Your Health (Get the book.)

"First discovered by a group of scientists at the Geron Corporation in Menlo Park, California, telomeres are sequences of nucleic acids extending from the ends of chromosomes. telomeres act to maintain the integrity of our chromosomes. Every time our cells divide, telomeres are shortened, leading to cellular damage and cellular death associated with aging. Scientists discovered that the key element in rebuilding our disappearing telomeres is the "immortalizing" enzyme telomerase—an enzyme found only in germ cells and cancer cells."
- Ronald Klatz and Robert Goldman, Stopping the Clock: Longevity for the New Millenium (Get the book.)

"They demonstrated a definitive connection between perceived and chronic stress and changes in telomeres, specific areas of the chromosome that are known to relate to the length of cell life and aging. It is critically important to human health that we shed our stigma about the nature of things emotional. Toughing things out, burying uncomfortable feelings, and living under stress can lead to physical pain and dysfunction and, most important, serious medical illness. The beauty of the TMS diagnosis is that it is a hopeful one that can result in a true cure."
- John E. Sarno, M.D., The Divided Mind: The Epidemic of Mindbody Disorders (Get the book.)

"Aging is essentially a process in which your cells lose their resilience; they lose their ability to repair damage because the things you might never have heard of (until now), like mitochondria and telomeres, aren't working the way they should. But it's within your power to boost that resilience and keep your vehicle going an extra couple hundred thousand miles. 3."
- Mehmet C. Oz., M.D. and Michael F. Roizen, M.D., You: Staying Young: The Owner's Manual for Extending Your Warranty (Get the book.)

"The Major Ager of shortened telomeres also slows down our stem cells, so the damage is compounded. The process of breathing is kind of like taking sandpaper to your lungs. But with fibrosis, the lungs are rough like cement. You can get the smoother, varnish-like lining you want by eliminating some of the risk factors, like smoking, high blood pressure, and diabetes."

- Mehmet C. Oz., M.D. and Michael F. Roizen, M.D., You: Staying Young: The Owner's Manual for Extending Your Warranty (Get the book.)

"When studies were carried out on mice, however, the theory didn't hold up: Strains of mice with long telomeres didn't live any longer than those with average-length telomeres. There are other holes in the argument that our telomeres decide our life span. While the regulation of telomerase may play some role in aging, the changes we associate with the "golden years"—muscle weakness, arthritis, heart disease, skin and hair changes, and declines in mental function—have been found to be unrelated to telomere shortening."
- David Heber, M.D., Ph.D., What Color is Your Diet? (Get the book.)

"Do you know if your telomeres are frayed?" "Meet me in the bedroom with the canola oil." After two minutes, open your eyes and let your partner know if you heard what was whispered. If not: You're showing signs of early hearing loss and should be especially aware of taking steps to prevent further damage. If so: Celebrate and proceed to the bedroom immediately. With canola oil. This test is easier if a man is the tester, due to the voice tones of most men-see page 297. Nervt I We > all know (and probably live with) people who have recently added some more words to their verbal repertoire."
- Mehmet C. Oz., M.D. and Michael F. Roizen, M.D., You: Staying Young: The Owner's Manual for Extending Your Warranty (Get the book.)

"The implication is that if you can reduce the effects that stress has on you, through such techniques as meditation (see page 350), you can increase your chance of rebuilding the telomeres and decrease the odds of having your cells die and contribute to age-related problems. Yes, you're stuck with the genes you were given, just as you're stuck with the decisions your parents made about where you grew up, and you can't return your genes for a complete refund. You can, however, change the way they function."

- Mehmet C. Oz., M.D. and Michael F. Roizen, M.D., You: Staying Young: The Owner's Manual for Extending Your Warranty (Get the book.)

"In order to prevent that information loss from making a difference, your chromosomes have what amounts to extra information at their tips; those bits of information are telomeres. Imagine you have a manuscript and need to make fifty copies but Kinko's has just thrown you a curveball. Instead of charging you money, they're just going to take one page off the end of your manuscript after every copy."
- Dr. Sharon Moalem, Survival of the Sickest: A Medical Maverick Discovers Why We Need Disease (Get the book.)

"When studies were carried out on mice, however, the theory didn't hold up: Strains of mice with long telomeres didn't live any longer than those with average-length telomeres. There are other holes in the argument that our telomeres decide our life span. While the regulation of telomerase may play some role in aging, the changes we associate with the "golden years"—muscle weakness, arthritis, heart disease, skin and hair changes, and declines in mental function—have been found to be unrelated to telomere shortening."
- David Heber, M.D., Ph.D., What Color is Your Diet? (Get the book.)

"But cancer cells can sometimes kick telomerase into high gear, so that the telomeres are replenished more rapidly. When that happens, there's less loss of genetic information, because the telomere buffer never runs out. The expiration date programmed into cells is canceled, and the cell can reproduce forever. When cancer cells are successful, it's usually with the help of telomerase. More than 90 percent of the cells in cancerous human tumors use telomerase."
- Dr. Sharon Moalem, Survival of the Sickest: A Medical Maverick Discovers Why We Need Disease (Get the book.)

"But once a cell replicates between fifty and sixty times, the telomeres are essentially gone and the good stuff is in jeopardy. Now, why would we evolve a limit against cellular reproduction? In a word? Cancer. if there's a health-related word more closely associated with fear and mortality than cancer, I don't know what it is. It's so widely assumed to be a likely death sentence that, in millions of families, it's barely spoken out loud; instead it's only spoken, if at all, in a kind of stage whisper. iiidLi i_ne."

- Dr. Sharon Moalem, Survival of the Sickest: A Medical Maverick Discovers Why We Need Disease (Get the book.)

"In 1986 Howard Cook and his co-workers discovered that germ cells have significantly longer telomeres than other cells. In addition to a lengthened telomere, germ cells contain an enzyme, telomerase, which continuously replaces the telomere fragment that is lost during cell division. No other cells possess this enzyme. Despite this extraordinary finding, it does not appear that aging and death result solely from telomere loss. Cells taken from a centenarian will still divide, yet they will reproduce fewer times than younger cells before finally dying."
- Russell L. Blaylock, M.D., Health and Nutrition Secrets (Get the book.)

"When a cell is young, it contains a chain of more than 1,000 telomeres. Each time the cell divides, it uses up ten or twenty telomeres from the chain. Eventually, the chain is devoured and the cell loses its ability to reproduce. The chain of telomeres in cancer cells is not exhausted. The length of the chain is thought to be protected by the enzyme telomerase. Scientists believe that telomerase enables the mutated cell to outwit death. Once the cells have been initiated with cancer, in order for the cancer to advance, it must be exposed to promoters."
- Laurie Deutsch Mozian, M.S., R.D., Foods That Fight Disease: A Simple Guide to Using and Understanding Phytonutrients to Protect and Enhance Your Health (Get the book.)

"Compared with normal children, the telomeres of progeria children at birth are like those of a ninety-year-old. In Werner's syndrome, telomeres are at normal length at birth but appear to shorten faster than normal telomeres. The key to extending human life span will ultimately involve preserving or restoring telomere length to the DNA. To read more about the telomere theory of aging, we refer you to Reversing Human Aging by Michael Fossel, Ph.D., M.D.10 The Free-Radical Theory The best damage theory is the free-radical theory of aging."
- Michael T. Murray, N.D., Joseph E. Pizzorno, N.D., Encyclopedia of Natural Medicine, Revised Second Edition (Get the book.)

"They pull off this feat by using telomerase to fix their telomeres the same way that some cancer cells do. You can see why scientists believe stem cells have such potential to cure disease and alleviate suffering—they have the potential to become anything and they never run out of steam. Many scientists believe cancer prevention is the "reason" cells have evolved with a limit on the number of times they can reproduce. The flip side to the Hayflick limit, of course—compromise, compromise—is aging. Once cells hit the limit, future reproductions don't really work and things start to break down."
- Dr. Sharon Moalem, Survival of the Sickest: A Medical Maverick Discovers Why We Need Disease (Get the book.)

"What scientists are seeing is that with each new cell division, the telomeres are shortened, and that after these structures shrink down to a certain size, cell division stops. In other words, telomeres are a kind of biological clock, with decreasing length indicating a decreasing amount of cell lifetime left. But what if we could replenish shrinking telomeres? A recent development leads some researchers to think that this might be possible."
- Gary Null, Gary Null's Power Aging (Get the book.)

"Proceedings of the National Academy of Sciences in December 2004 indicated that major life stresses can actually damage the telomeres (the sections of DNA at the tips of chromosomes) inside the body's immune cells, decreasing the cells' lives. The study compared a group of women caring for children suffering from serious chronic conditions to a group of women with healthy children. An interesting feature of the study is that the results were strongly related to the perception of emotional stress."
- Margaret Ruby, The DNA of Healing: A Five-Step Process for Total Wellness and Abundance (Get the book.)

"Women in both groups who felt they were undergoing the highest stress levels had telomeres comparable to someone ten years older than they were. In a Washington Post article on this important finding, Dr. Dennis Novack of Drexel University College of Medicine said the new study showed that mind and body are not separate, that "the very molecules in our bodies are responsive to our psychological environment."5 While more research is still needed, the study does point to a direct relationship not just between chronic stress and our health but between stress (or emotions) and our genes."

- Margaret Ruby, The DNA of Healing: A Five-Step Process for Total Wellness and Abundance (Get the book.)

"In other words, telomeres are a kind of biological clock, with decreasing length indicating a decreasing amount of cell lifetime left. But what if we could replenish shrinking telomeres? A recent development leads some researchers to think that this might be possible. A naturally occurring enzyme called telomerase has been discovered, and what it seems able to do is copy the telomeres' RNA in the form of DNA, and then put it on the ends of chromosomes, thereby extending cell life. The down side of telomerase is that it plays a role in the unchecked cell growth that is cancer."
- Gary Null, Gary Null's Power Aging (Get the book.)

"Every time our cells divide, telomeres are shortened, leading to cellular damage and cellular death associated with aging. Scientists discovered that the key element in rebuilding our disappearing telomeres is the "immortalizing" enzyme telomerase—an enzyme found only in germ cells and cancer cells. Telomerase appears to repair and replace telomeres, manipulating the "clocking" mechanism that controls the lifespan of dividing cells. Future development of a telomerase inhibitor may be able to cease cancer cells from dividing and presumably convert them back into normal cells."
- Ronald Klatz and Robert Goldman, Stopping the Clock: Longevity for the New Millenium (Get the book.)

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